Research reveals how aging cells can be an underlying cause of kidney damage
A study in mice has found that stress and tissue damage initiated by angiotensin II, a molecule that is known to increase blood pressure and stiffening in the linings of blood vessels, leads to cellular senescence, a process by which a cell ages and permanently stops dividing but does not die. Importantly, when the researchers eliminated senescent cells from the mice, tissues returned to a normal state in spite of a continued infusion of angiotensin II.
Materials provided by Georgetown University Medical Center. Note: Content may be edited for style and length.
Source link aaaaa